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bioRxiv · 10.1101/2025.05.15.654308

Redirecting cytomegalovirus immunity against pancreas cancer for immunotherapy

Abstract

Immunotherapy shows limited success in pancreatic cancer, largely due to a low mutational burden and immunosuppressive microenvironment. Here we hypothesized that pre-existing antiviral immunity can be redirected to control pancreatic tumors. Cytomegalovirus (CMV, a {beta}-herpesvirus) was chosen, as the majority of the population is infected and it induces an extremely large/broad memory T cell response. Mice latently infected with murine CMV (MCMV) were orthotopically implanted with pancreatic cancer cells and treated with systemic injections of MCMV T-cell epitopes. The therapy promoted preferential accumulation of MCMV-specific T cells within pancreatic tumors, delaying tumor growth and increasing survival. Immunophenotyping and scRNAseq analyses showed these T cells were highly activated and cytotoxic, leading to increased tumor necrosis and caspase-3 activation. Finally, therapy was enhanced when combined with subtherapeutic doses of gemcitabine chemotherapy. Together, these results show that CMV-specific T cells can be repurposed to combat pancreatic cancer. SignificanceOur studies reveal that CMV-specific viral memory T cells can be re-directed to control a solid tumor normally refractory to immunotherapy via a simple, intravenous injection of T cell peptide epitopes. This mutation agnostic approach has significant potential for the development of "off-the-shelf" therapeutics by stimulating pre-existing antiviral memory and it is widely applicable due to the high prevalence of CMV.

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BibTeXRIS

Marrocco, R., Patel, J., Medari, R., Salu, P., Lucero-Meza, E., Brunel, S., Martsinkovskiy, A., Sun, S., Gulay, K., Jaljuli, M., Mose, E., Lowy, A., Benedict, C. A., Hurtado de Mendoza, T.. 2025-05-19. Redirecting cytomegalovirus immunity against pancreas cancer for immunotherapy. https://doi.org/10.1101/2025.05.15.654308

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