Search bioRxiv⌕ Search

bioRxiv · 10.64898/2026.10.02.756323

Learning Interpretable Switching Dynamics in Shared Neural-Behavioral Latent Space

Abstract

Modern recording technologies enable simultaneous measurement of high-dimensional neural activity and rich behavioral variables, creating both an opportunity and a modeling challenge: identifying the shared latent structure that mediates the brain-behavior relationship and the underlying dynamics through which it evolves across behavioral states. Existing approaches focus on different parts of this challenge: shared representation learning methods identify neural-behavioral subspaces but typically do not model dynamical structure, while dynamical models capture temporal variation and discrete state transitions in neural activity, but do not explicitly disentangle shared neural-behavioral structure from modality-specific variability. We introduce Switching Shared Latent Dynamics (SSLD), a unified framework that learns a latent representation shared between neural activity and behavior and endows this shared space with nonlinear switching recurrent dynamics to reflect the hypothesis that behaviorally relevant neural representations exhibit structured dynamical changes across behavioral states. Complementary private latent variables capture modality-specific variability, providing a window into neural activity that does not directly interact with behavior. We evaluate SSLD on a simulated dataset and four diverse experimental datasets spanning species, brain regions, and recording modalities: monkey motor and premotor cortex during reaching, somatosensory cortex during a bump task, widefield calcium imaging across mouse dorsal cortex during (a) self-initiated decision-making and (b) spontaneous movements. Across all four datasets, SSLD accurately reconstructs neural and behavioral signals, recovers discrete states in the dynamics that align with experimentally defined behavioral epochs, and isolates behaviorally-relevant neural information in the shared latent while preserving private neural variability. Ablations confirm that shared representation learning, behavioral supervision, and switching dynamics each contribute to performance. SSLD offers an interpretable approach to modeling shared neural-behavioral dynamics that complements ongoing efforts toward foundation models for neuroscience.

Explore related subjects

Keep this discovery

Explore connections, maps & timelines

BibTeXRIS

Zhang, Y., Caro, J. O., Oren, R., Higley, M. J., Cardin, J., Churchland, A., Saxena, S.. 2026-10-09. Learning Interpretable Switching Dynamics in Shared Neural-Behavioral Latent Space. https://doi.org/10.64898/2026.10.02.756323

Cite the original work for its findings. Save a collection to share your selection of sources.

KEEP EXPLORING

Related preprints

Brain representations of visual short-term memories during healthy human aging: occipital decline and frontoparietal compensation

Visual short-term memory declines steeply with age, but the link to neural representations of memory content remains unknown. We used fMRI decoding to relate remembered motion-direction representations in visual and frontoparietal cortex to age and performance, in 115 adults, aged 25-87, from the Cambridge Centre for Ageing and Neuroscience (Cam-CAN) cohort. Decoding was strongest in visual cortex, but also significant in frontoparietal regions, with different profiles. Visual representations weakened with memory load and age, and statistically mediated age-related performance decline, especially during recall. This coupling, however, weakened with age: some older adults retained stronger visual representations than performance implied, suggesting impaired access to a partially preserved memory trace. Frontoparietal representations, instead, were largely insensitive to load and strengthened with age during maintenance, partially offsetting performance decline. Therefore, interventions to boost VSTM in older adults might target compensatory frontoparietal representations during memory maintenance, and readout of untapped visual representations at recall.

neuroscience↗

Lexical surprisal drives pupil dilation during continuous story listening

The pupil famously adjusts its dilation in response to light, but its role extends far beyond this basic sensory function. Its fluctuations index arousal and mental effort, beyond luminance-related dynamics, making it a sensitive marker of internal cognitive states. A growing body of work shows that these pupil-linked signals extend beyond the visual domain. However, which processes drive pupil responses in more naturalistic settings remains unclear as most evidence comes from tightly controlled paradigms that strip away the richness of real-world experience. Language offers a uniquely powerful window into this question, as it can allow us to create rich paradigms without having any visual, i.e. luminance, confounds. Moreover, language comprehension hinges on continuous prediction: listeners constantly generate expectations about upcoming input, and the discrepancy between expectation and actual input - contextual prediction error, or surprisal - carries measurable cognitive costs. Here, we tested the hypothesis that pupil-linked arousal indexes these linguistic surprisal signals during naturalistic listening. We recorded pupil responses while volunteers listened to naturalistic spoken stories in darkness, which were either intact or reversed. Reversed speech preserves the acoustics of the signal while destroying its intelligibility. Pupil dilation selectively tracked lexical surprisal within a late window of 1,700 to 2,350 milliseconds following word onset, in forward, but not control, backward speech. These findings indicate that pupil-linked arousal is tuned to global, context-dependent processing, tracking the cognitive cost of updating a predictive model during naturalistic listening.

neuroscience↗

Disrupting SORL1-retromer endosomal recycling in mice drives the age-dependent neurodegenerative signature of Alzheimer's disease

MRI studies have mapped the atrophic signature that distinguishes Alzheimer's disease (AD), isolating regions differentially vulnerable to its age-dependent neurodegenerative process. We test the hypothesis that an endosomal recycling pathway regulated by the interaction of SORL1 with retromer, which is pathogenically disrupted in AD, might drive the neurodegenerative process via multiple yearlong studies in over 140 wildtype and SORL1 deficient mice, modeling AD's causative mutations. MRI studies revealed that SORL1 deficiency induced an age-dependent AD-pattern of atrophy, centered on the trans-entorhinal cortex, and that atrophy was prevented by SORL1 restoration, secondary to overexpressing the retromer protein VPS26b. Postmortem studies validated that the atrophy reflects neuronal loss. Proteomic profiling revealed that the atrophy is associated with reductions in cell death suppressors, one of which was then found deficient in the trans-entorhinal cortex of AD patients. Besides supporting the hypothesis, taken together with other findings, the results suggest that SORL1-retromer disruptions represent a therapeutically-amenable common 'neurodegeneration pathway' in AD.

neuroscience↗