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bioRxiv · 10.64898/2026.07.23.740218

Spatial control of mitochondrial retrograde signaling by nuclear pore-associated contact sites

Abstract

Mitochondria relay their functional state to the nucleus via retrograde signaling, yet whether the spatial organization of the mitochondrial network plays a role in this process remains unclear. Here, we show that stress-induced clustering of mitochondria around the nucleus is a crucial part of the retrograde response. Perinuclear clustering facilitates the formation of mitochondria- nucleus contact sites (MNCS) and the nuclear entry of GPS2, a key mediator of mitochondrial retrograde signaling essential for activating nuclear-encoded mitochondrial and stress-response genes in response to various mitochondrial stressors. Unexpectedly, TSPO-driven MNCS are dispensable for GPS2-based retrograde signaling. Instead, we identify the mitochondrial import receptor TOMM70 and the nucleoporin RanBP2/NUP358 as components of a stress-induced nuclear pore-associated tethering complex required for promoting GPS2 nuclear translocation and activation of downstream programs. These findings establish MNCS as a functional gateway for mitochondrial retrograde signaling, highlighting that organelle positioning and tethering at the nuclear pore provide an unexpected layer of stress regulation.

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BibTeXRIS

Mitra, S., Burrone, G., Rubarajan, S., Klein, C., Kwok, T., Gutta, S., Li, K., Berson, E., Orofino, J., Cardamone, M. D., Blower, M. D., Perissi, V.. 2026-07-24. Spatial control of mitochondrial retrograde signaling by nuclear pore-associated contact sites. https://doi.org/10.64898/2026.07.23.740218

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