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bioRxiv · 10.64898/2026.03.05.709908

Apical spectrin organizes cortical actin filament bundles to pattern C. elegans cuticle ridges

Abstract

The apical extracellular matrix can form elaborate three-dimensional structures on animal surfaces. To better understand the mechanisms that pattern and shape these structures, we focus on development of collagen-rich cuticle ridges (alae) in adult C. elegans. Previous studies suggested that longitudinal actin filament bundles (AFBs) in the lateral seam epidermis specify alae position through a mechanism that involves post-secretory matrix delamination. Here we identify additional components of this highly organized cortical actin network and show that loss of the apical {beta}H-spectrin SMA-1 specifically disrupts organization of the two AFBs that would normally flank the site where the middle alae ridge forms. Correspondingly, sma-1 loss, or mutation of its actin binding domains, also disrupts formation of the middle alae ridge. Ultrastructurally, sma-1 mutants have expanded regions of matrix delamination that can explain middle ridge loss. Together, these data highlight the importance of apical spectrin for organizing a patterned actin network within epithelia and show that, via its effects on actin organization, spectrin can also change the extracellular matrix and its patterns on animal surfaces.

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Sarwar, P. F., Barker, T. J., Nguyen, K. C. Q., Chan, F.-Y., Hall, D. H., Carvalho, A. X., Sundaram, M. V.. 2026-03-09. Apical spectrin organizes cortical actin filament bundles to pattern C. elegans cuticle ridges. https://doi.org/10.64898/2026.03.05.709908

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