Search bioRxiv⌕ Search

bioRxiv · 10.1101/284562

A probabilistic interpretation of PID controllers using active inference

Abstract

In the past few decades, probabilistic interpretations of brain functions have become widespread in cognitive science and neuroscience. The Bayesian brain hypothesis, predictive coding, the free energy principle and active inference are increasingly popular theories of cognitive functions that claim to unify understandings of life and cognition within general mathematical frameworks derived from information theory, statistical physics and machine learning. Furthermore, it has been argued that one such proposal, active inference, combines both information and control theory and has its roots in cybernetics studies of the brain. The connections between information and control theory have been discussed since the 1950s by scientists like Shannon and Kalman and have recently risen to prominence in modern stochastic optimal control theory. How-ever, the implications of the confluence of these two theoretical frame-works for the biological sciences have been slow to emerge. Here we argue that if the active inference proposal is to be taken as a general process theory for biological systems, we need to consider how existing control theoretical approaches to biological systems relate to it. In this work we will focus on PID (Proportional-Integral-Derivative) controllers, one of the most common types of regulators employed in engineering and more recently used to explain behaviour in biological systems, e.g. chemotaxis in bacteria and amoebae or robust adaptation in biochemical networks. Using active inference, we derive a probabilistic interpretation of PID controllers, showing how they can fit a more general theory of life and cognition under the principle of (variational) free energy minimisation once we use only simple linear generative models.

Explore related subjects

Keep this discovery

Explore connections, maps & timelines

BibTeXRIS

Baltieri, M., Buckley, C. L.. 2018-03-19. A probabilistic interpretation of PID controllers using active inference. https://doi.org/10.1101/284562

Cite the original work for its findings. Save a collection to share your selection of sources.

KEEP EXPLORING

Related preprints

Stochastic and Heritable Responses in Cell-Size Regulation and Survival to pH Stress in C. glutamicum

Bacterial survival in fluctuating environments depends on the ability of individual cells to maintain physiological homeostasis under stress, yet how single cells recover from transient pH fluctuations remains poorly understood. We tracked the cell-size dynamics and survival of Corynebacterium glutamicum cells exposed to short, strong acid (pH 4, 20 min) or alkaline (pH 11, 15 min) pulses. Acid stress considerably increases the size added during the stress cycle, whereas alkaline stress increases it only modestly. Nevertheless, surviving cells maintain their division strategy and restore size homeostasis in the next cycle. Coordination between relatives is short-lived: sister cells have similar cell-cycle durations after stress, but durations become uncorrelated once the last division shared by two cells precedes the stress by more than about 1.5 generations. Survival of acid stress favors larger cells and is largely stochastic, with no detectable lineage heritability. In contrast, survival of alkaline stress favors smaller cells and is transiently heritable: co-survival probability remains high among relatives separated by up to two generations. These findings provide insight into pH survival in C. glutamicum and present statistical methods to study cell-size regulation and survival under transient, severe environmental fluctuations.

systems biology↗

Enzyme-Constrained Metabolic Modeling Reveals CO2-Dependent Growth Control in Picochlorum celeri

Picochlorum celeri grows 11.6 times faster at elevated CO2 than in air, yet the abundance of its metabolic enzymes is only 17% higher. To explain this, we reconstructed iPiccel954, a genome-scale metabolic model with 3525 reactions, 2973 metabolites and 954 genes, and eciPiccel954, its enzyme-constrained version limited by the measured enzyme abundance in air and at elevated CO2. Calibrating the enzyme-constrained models against the measured proteome showed that a similar protein budget supports very different growth depending on how it is allocated. With urea as the nitrogen source, urea carboxylase was the only step whose relaxation alone raised growth, by 627% in air and 78% at elevated CO2. Bottleneck analysis showed that growth is limited by many enzymes spread across the network in both conditions, that the limiting enzymes differ between the two, and that control over starch metabolism seen in air disappears at elevated CO2. Measured proteomes agreed with these predictions, since the predicted bottleneck enzymes were more abundant than other enzymes. Rapid growth in P. celeri therefore reflects how the protein budget is allocated more than how large it is. Therefore, eciPiccel954 provides a quantitative basis for engineering this alga for biotechnological application.

systems biology↗

Organelle Aging Predicts Human Disease

Aging affects cellular organelles differently, but whether subcellular aging predicts human disease remains unknown. Here, we integrated organelle-enriched proteins in plasma and cerebrospinal fluid proteomes from 61,931 individuals to model aging of 10 organelles across the human lifespan. Organelle aging was heterogeneous with lysosomes, plasma membranes, and Golgi compartments accelerating rapidly after age 60. Individuals with four or more prematurely aged compartments exhibited up to 22-fold higher kidney disease risk and up to 8-fold higher mortality risk. Major diseases are characterized by distinct organelle aging signatures and disease risk increased with the number of accelerated organelles. APOE-{varepsilon}4 dosage was associated with lysosomal and mitochondrial aging and reduced PDP1 levels, implicating altered energy metabolism. Decomposition of organelle signature coefficients into biological programs further showed that diseases are associated with distinct subcellular patterns. These findings establish organelle-resolved aging as a measurable dimension of human biology, revealing subcellular mechanisms underlying disease susceptibility and resilience.

systems biology↗