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bioRxiv · 10.1101/2025.07.21.666029

Immunodominance is a poor predictor of vaccine-induced T follicular helper cell quality

Abstract

Rational engineering of vaccine immunogens to focus B cell responses on potently neutralizing epitopes is a promising approach to improve the potency, breadth and durability of viral vaccines. Such strategies, however, can compromise vaccine immunogenicity through the unintended exclusion of CD4+ T cell epitopes, which are critical for the development of T follicular helper (TFH) cells and to support high affinity antibody production. Using a prototypic influenza HA stem immunogen lacking effective CD4+ T cell help in BL6 mice, we interrogated the minimal requirements for T cell help needed to drive serological responses to vaccination. We find that priming of naive CD4 T cells is markedly efficient, however the immunodominance of a given CD4 T cell epitope is not predictive of the propensity to provide high quality help to antigen-specific B cells. In the context of soluble antigens, provision of a single MHC class II epitope is sufficient to drive robust germinal centre responses and serum IgG titres. However not all CD4 epitopes provide equivalent levels of B cell help, despite priming comparable numbers of antigen-specific CD4 T cells. Finally, we show multimerizing and arraying antigens on nanoparticle scaffolds unlocks highly subdominant, near-undetectable CD4 T cell helper responses to support a T-dependent antibody response. Our findings emphasize the importance of CD4+ T cell help for programing robust and durable humoral immunity, and provide crucial insights to guide the rational incorporation of favorable T cell epitopes into vaccines.

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BibTeXRIS

Tan, H.-X., Zheng, M. Z. M., Wragg, K., Murdiyarso, L., Pilapitiya, D., Kelly, A., Esterbauer, R., Gonelli, C. A., Wheatley, A. K., Juno, J. A.. 2025-07-21. Immunodominance is a poor predictor of vaccine-induced T follicular helper cell quality. https://doi.org/10.1101/2025.07.21.666029

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