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bioRxiv · 10.1101/2025.07.02.662713

A FRET-based high-throughput screening assay for the discovery of Mycobacterium tuberculosis DNA ADP-ribosylglycohydrolase DarG inhibitors

Abstract

DarTG2 is a conserved toxin-antitoxin ADP-ribosylation system that regulates bacterial survival and the anti-phage response found in many pathogenic bacteria, including Mycobacterium tuberculosis. While DNA ADP-ribosyltransferase (DarT2) toxin mono-ADP-ribosylates a single- stranded DNA sequence motif and potentially induces bacterial dormancy, its cognate DNA ADP- ribosylglycohydrolase (DarG) antitoxin reverses the modification and restores bacterial growth. Therefore, developing DarG selective inhibitors may represent a promising and novel strategy to combat drug-resistant tuberculosis. However, no small molecule inhibitors targeting DarG have been identified to date and establishing a high-throughput screening assay based on its DNA ADP- ribosylhydrolase activity would be challenging. Here, we developed and optimized a simple and robust fluorescence resonance energy transfer (FRET)-based high-throughput screening assay to identify small molecule inhibitors targeting DarG macrodomain in M. tuberculosis. We generated a FRET pair of M. tuberculosis DarG macrodomain and poly-ADP-ribosylated peptide fused with compatible fluorophores. Screening the target-focused phenotypic library using this method led to the identification of pranlukast, which selectively inhibited the DNA ADP-ribosylhydrolase activity of DarG in M. tuberculosis and its orthologues without affecting human mono-ADP- ribosyl binders and erasers. Since pranlukast has previously been reported to reduce M. tuberculosis burden, further investigation into its action mechanism in this context would be valuable.

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BibTeXRIS

Duong, M. T. H., Lu, Y., Ahel, I., Lehtiö, L.. 2025-07-05. A FRET-based high-throughput screening assay for the discovery of Mycobacterium tuberculosis DNA ADP-ribosylglycohydrolase DarG inhibitors. https://doi.org/10.1101/2025.07.02.662713

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