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bioRxiv · 10.1101/2024.06.24.600538

Structural and mechanistic studies on human LONP1 redefine the hand-over-hand translocation mechanism

Abstract

AAA+ enzymes use energy from ATP hydrolysis to remodel diverse cellular targets. Structures of substrate-bound AAA+ complexes suggest that these enzymes employ a conserved hand-over-hand mechanism to thread substrates through their central pore. However, the fundamental aspects of the mechanisms governing motor function and substrate processing within specific AAA+ families remain unresolved. We used cryo-electron microscopy to structurally interrogate reaction intermediates from in vitro biochemical assays to inform the underlying regulatory mechanisms of the human mitochondrial AAA+ protease, LONP1. Our results demonstrate that substrate binding allosterically regulates proteolytic activity, and that LONP1 can adopt a configuration conducive to substrate translocation even when the ATPases are bound to ADP. These results challenge the conventional understanding of the hand-over-hand translocation mechanism, giving rise to an alternative model that aligns more closely with biochemical and biophysical data on related enzymes like ClpX, ClpA, the 26S proteasome, and Lon protease.

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BibTeXRIS

Mindrebo, J. T., Lander, G. C.. 2024-06-25. Structural and mechanistic studies on human LONP1 redefine the hand-over-hand translocation mechanism. https://doi.org/10.1101/2024.06.24.600538

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