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bioRxiv · 10.1101/2020.06.05.127290

Cold induced chromatin compaction and nuclear retention of clock mRNAs resets the circadian rhythm

Abstract

Cooling patients to sub-physiological temperatures is an integral part of modern medicine. We show that cold exposure induces temperature-specific changes to the higher-order chromatin and gene expression profiles of human cells. These changes are particularly dramatic at 18{degrees}C, a temperature synonymous with that experienced by patients undergoing controlled deep-hypothermia during surgery. Cells exposed to 18{degrees}C exhibit largely nuclear-restricted transcriptome changes. These include the nuclear accumulation of core circadian clock suppressor gene transcripts, most notably REV-ERB. This response is accompanied by compaction of higher-order chromatin and hindrance of mRNPs from engaging nuclear pores. Rewarming reverses chromatin compaction and releases the transcripts into the cytoplasm, triggering a pulse of suppressor gene proteins that resets the circadian clock. We show that cold-induced upregulation of REV-ERB alone is sufficient to trigger this resetting. Our findings uncover principles of the cellular cold-response that must be considered for current and future applications involving therapeutic deep-hypothermia.

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Fischl, H., McManus, D., Oldenkamp, R., Schermelleh, L., Mellor, J., Jagannath, A., Furger, A.. 2020-06-06. Cold induced chromatin compaction and nuclear retention of clock mRNAs resets the circadian rhythm. https://doi.org/10.1101/2020.06.05.127290

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