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bioRxiv · 10.1101/2020.01.21.914127

Membrane-mediated ligand unbinding of the PK-11195 ligand from the translocator protein (TSPO)

Abstract

The translocator protein (TSPO), previously known as the peripheral benzodiazepine receptor, is of longstanding medical interest as both a biomarker for neuroinjury and a potential drug target for neuroinflammation and other disorders. Recently it was shown that ligand residence time is a key factor determining steroidogenic efficacy of TSPO-binding compounds. This spurs interest in simulations of (un)binding pathways of TSPO ligands, which could reveal the molecular interactions governing ligand residence time. In this study, we use a weighted ensemble algorithm to determine the unbinding pathway for different poses of PK-11195, a TSPO ligand used in neuroimaging. In contrast with previous studies, our results show that PK-11195 does not dissociate directly into the solvent but instead dissociates via the lipid membrane by going between the transmembrane helices. We analyze this path ensemble in detail, constructing descriptors that can facilitate a general understanding of membrane-mediated ligand binding. We construct a Markov state model using additional straightforward simulations to determine pose stability and kinetics of ligand unbinding. Together we combine over 40 {micro}s of trajectory data to form a coherent picture of the ligand binding landscape. We find that all poses are able to interconvert before unbinding, leading to single mean first passage time estimate for all starting poses which roughly agrees with the experimental quantity. The ligand binding transition state predicted by our combined model occurs when PK-11195 is already in the membrane and does not involve direct ligand-protein interactions. This has implications for the design of new long residence-time TSPO ligands. SIGNIFICANCEKinetics-oriented drug design is an emerging objective in drug discovery. However, while ligand binding affinity (or the binding free energy) is purely a function of the bound and unbound states, the binding kinetics depends on the nature of the paths by which the (un)binding occurs. This underscores the importance of approaches that can reveal information about the ensemble of (un)binding paths. Here we used advanced molecular dynamics approaches to study the unbinding of PK-11195 from TSPO and find it dissociates from the protein by dissolving into the membrane, and that the transition state occurs after the PK-11195 molecule has already separated from TSPO. These results motivate the design of future long-residence time TSPO ligands that destabilize the membrane-solvated transition state.

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BibTeXRIS

Dixon, T., Uyar, A., Ferguson-Miller, S., Dickson, A.. 2020-01-21. Membrane-mediated ligand unbinding of the PK-11195 ligand from the translocator protein (TSPO). https://doi.org/10.1101/2020.01.21.914127

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