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Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells
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Based on 525 indexed works selected for this snapshot; the candidate window is limited. Counts describe this index, not the complete source archives. Prepared from the PostgreSQL corpus; source versions are checked before display. Snapshot 2026-11-07. Up to 32 works or names per graph.
Subjects & research connections
Connections use shared source subjects, names, places, and explicitly mentioned entities. A shared label is not evidence of a citation, experimental result, or verified species identification.
Relationships as a list (31)
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells Preprint · Wang, Q. 2. A biomimetic SPARC platform coupling adaptor recruitment and BRS-Lck condensation for enhanced NK/T cell combination therapy
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells molecular biology · Preprint 3. T cell transcriptomes in simulated microgravity in vivo
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells molecular biology · Preprint 4. Single-probe, high-yield spatial mapping of diverse short RNA motifs
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells molecular biology · Preprint 5. Comparative profiling of sperm small RNAs in cnidarians highlights potential for paternal inheritance
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells molecular biology · Preprint 6. Aging activates latent enhancers that can be reversed withexercise
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells molecular biology · Preprint 7. An orphan LuxR regulator controls plant developmental reprogramming during interaction with Novosphingobium resinovorum HR1a
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells molecular biology · Preprint 8. Physiological mapping and prediction reveal stress-responsive translation initiation landscapes
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells molecular biology · Preprint 9. Optimal compositions of potent sliced-siRNA with DNA bases substitution and insights of Ago2 slicing actions
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells molecular biology · Preprint 10. HiSpoT: a novel one-tube dual-barcode PCR method for NGS spoligotyping amplicon library preparation in Mycobacterium tuberculosis complex
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells molecular biology · Preprint 11. Genomic features and instability of linezolid resistance in vancomycin-resistant Enterococcus faecalis ST525
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells molecular biology · Preprint 12. Global repression of mesenchymal genes in epithelial cells by the histone demethylase LSD1
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells molecular biology · Preprint 13. The long noncoding RNA FOSIL shapes the dynamics of FOS induction
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells molecular biology · Preprint 14. Prokaryotic homologs of the major vault protein form engineerable vault nanocapsules
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells molecular biology · Preprint 15. Optogenetic induction of TAD boundary pairing promotes gene activity
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells molecular biology · Preprint 16. Hijacking Cellular S-Acylation with Molecular Assemblies Rewires Oncogenic Signaling
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells molecular biology · Preprint 17. PARP1-mediated PARylation of SPRTN restrains its DNA-protein crosslink repair activity
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells molecular biology · Preprint 18. Metformin lowers HMGA1a and RBM3 protein in breast cancer cells, and its effect on ESR1 splicing depends on cell density and exposure time
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells molecular biology · Preprint 19. The mycotoxin zearalenone couples secondary metabolism to N-acetylglucosamine catabolism
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells molecular biology · Preprint 20. 2'-O-Methylated ncRNA condensates form a recombination center for IgH synapsis
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells molecular biology · Preprint 21. Stepwise assembly and mechanical stability of nsP3 Helical Scaffolds in Chikungunya Virus
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells molecular biology · Preprint 22. Stoichiometric Encoding of Amplification Kinetics Enables High-Level Multiplexing in Single-Channel TaqMan Real-Time PCR
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells Preprint 23. Integrated Biomedical System
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells Preprint 24. Expression of ACE2 and TMPRSS2, the SARS2-CoV-2 receptor and co-receptor, in prostate epithelial cells
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells Preprint 25. Where and how does d-amphetamine act to reveal antipsychotic-induced dopamine supersensitivity in rats?
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells Preprint 26. Irradiated mesenchymal stromal cells induce genetic instability in human CD34+ cells
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells Preprint 27. IFT20 is critical for early chondrogenesis during endochondral ossification
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells Preprint 28. Combining genetic resources and elite material populations to improve the accuracy of genomic prediction in apple
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells Preprint 29. TGF-beta1 induced S100 family protein expression is associated with epithelial to mesenchymal transition states and poor survival in pancreatic cancer
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells Preprint 30. DOTA chelation through click chemistry enables favorable biodistribution of 89Zr-radiolabeled antibodies: A comparison with DFO chelation
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells Preprint 31. The type of DNA damage response after Decitabine treatment depends on the level of DNMT activity
- 1. Inactive USP14 and inactive UCHL5 cause accumulation of distinct ubiquitinated proteins in mammalian cells Preprint 32. Alteration of mechanical stresses in the murine brain by age and hemorrhagic stroke
Citation graph
Arrows run from the citing work to its reference. Only explicit source/provider reference lists are used. Incoming links cover this candidate window; this is not a global citation count.
No supported relationships are available in this snapshot. This does not mean that no relationships exist.
Collaborator network
Shared authorship within up to 200 candidate works (2 examined); up to 32 names shown. Names are matched as supplied, without verified person disambiguation. Shared credit does not necessarily establish personal collaboration.
Relationships as a list (26)
- Chadchankar, J. 1 shared work Korboukh, V.
- Chadchankar, J. 1 shared work Doig, P.
- Chadchankar, J. 1 shared work Jacobsen, S.
- Brandon, N. 1 shared work Chadchankar, J.
- Chadchankar, J. 1 shared work Moss, S.
- Chadchankar, J. 1 shared work Wang, Q.
- Doig, P. 1 shared work Korboukh, V.
- Jacobsen, S. 1 shared work Korboukh, V.
- Brandon, N. 1 shared work Korboukh, V.
- Korboukh, V. 1 shared work Moss, S.
- Korboukh, V. 1 shared work Wang, Q.
- Doig, P. 1 shared work Jacobsen, S.
- Brandon, N. 1 shared work Doig, P.
- Doig, P. 1 shared work Moss, S.
- Doig, P. 1 shared work Wang, Q.
- Brandon, N. 1 shared work Jacobsen, S.
- Jacobsen, S. 1 shared work Moss, S.
- Jacobsen, S. 1 shared work Wang, Q.
- Brandon, N. 1 shared work Moss, S.
- Brandon, N. 1 shared work Wang, Q.
- Moss, S. 1 shared work Wang, Q.
- Wang, Q. 1 shared work Wu, S.
- Qiu, Y. 1 shared work Wang, Q.
- Lou, J. 1 shared work Wang, Q.
- Lou, C. 1 shared work Wang, Q.
- Chen, H. 1 shared work Wang, Q.
Publication timeline
Publication years for these 32 related works; 0 have no source publication date. This is a discovery sample, not a measure of research output or growth.
2016 · 1 work
2018 · 1 work
2020 · 3 works
2021 · 2 works
2022 · 2 works
2023 · 2 works
2026 · 21 works
- A biomimetic SPARC platform coupling adaptor recruitment and BRS-Lck condensation for enhanced NK/T cell combination therapy
- T cell transcriptomes in simulated microgravity in vivo
- Single-probe, high-yield spatial mapping of diverse short RNA motifs
- Comparative profiling of sperm small RNAs in cnidarians highlights potential for paternal inheritance
- Aging activates latent enhancers that can be reversed withexercise
- An orphan LuxR regulator controls plant developmental reprogramming during interaction with Novosphingobium resinovorum HR1a
- Physiological mapping and prediction reveal stress-responsive translation initiation landscapes
- Optimal compositions of potent sliced-siRNA with DNA bases substitution and insights of Ago2 slicing actions
- HiSpoT: a novel one-tube dual-barcode PCR method for NGS spoligotyping amplicon library preparation in Mycobacterium tuberculosis complex
- Genomic features and instability of linezolid resistance in vancomycin-resistant Enterococcus faecalis ST525
- Global repression of mesenchymal genes in epithelial cells by the histone demethylase LSD1
- The long noncoding RNA FOSIL shapes the dynamics of FOS induction
- Prokaryotic homologs of the major vault protein form engineerable vault nanocapsules
- Optogenetic induction of TAD boundary pairing promotes gene activity
- Hijacking Cellular S-Acylation with Molecular Assemblies Rewires Oncogenic Signaling
- PARP1-mediated PARylation of SPRTN restrains its DNA-protein crosslink repair activity
- Metformin lowers HMGA1a and RBM3 protein in breast cancer cells, and its effect on ESR1 splicing depends on cell density and exposure time
- The mycotoxin zearalenone couples secondary metabolism to N-acetylglucosamine catabolism
- 2'-O-Methylated ncRNA condensates form a recombination center for IgH synapsis
- Stepwise assembly and mechanical stability of nsP3 Helical Scaffolds in Chikungunya Virus
- Stoichiometric Encoding of Amplification Kinetics Enables High-Level Multiplexing in Single-Channel TaqMan Real-Time PCR
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