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Biology subjects

Yu, C.-Y.

Publications and source records attributed to Yu, C.-Y..

2 recordsLinked to original sources

Glycosylation-dependent sorting of an Arabinogalactan protein SLEEPING BEAUTY mediates apical tip growth and osmosensing in Physcomitrium patens

Polarized tip growth is a key morphological adaption that facilitated land plant terrestrialization, and arabinogalactan proteins (AGPs) are central regulators of this process. However, how glycosylation modulates AGP function during tip growth remains poorly understood. Here, we show that the AGP SLEEPING BEAUTY (SB) is required for cell wall integrity during protonemal tip growth in the moss Physcomitrium patens, a function that is conserved in the distantly related vascular plant Arabidopsis thaliana. Loss of SB function disrupts cellulose microfibril organization and compromises cell wall integrity, leading to altered tip growth dynamics and geometry. Using a hypoglycosylated SB variant, we demonstrate that glycosylation of Pro55, Pro92, and Pro94 within a highly intrinsically disordered region is required for proper SB secretion and vacuolar trafficking; loss of these modifications results in pronounced inhibition of tip growth. We propose that glycosylation of the intrinsically disordered domain in AGP fine-tunes SB abundance at the cell surface, thereby facilitating cellulose microfibril biosynthesis and/or assembly to regulate robust polarized tip growth.

plant biology↗

Astrocyte-specific secretome profiling reveals its correlation with neurological disorders

Secreted proteins mediate intercellular communication throughout the lives of multicellular organisms. However, due to the lack of new technology for secreted protein capturing, the progress of "secretomics lags behind. Here, we report a two-step secretome enrichment method (tsSEM) combining unnatural amino acid labeling and click chemistry-based biorthogonal reaction, which enables in vitro secretome profiling in the presence of serum. Using this novel method, we systematically investigated the secretome of human iPSCs-derived astrocytes (iAst) in different disease models and identified a panel of astrocyte-secreted proteins that are responsible for its non-cell autonomous toxicity under disease conditions. Furthermore, we validated two astrocytes-derived novel neurotrophic proteins, FAM3C and KITLG, which we identified from disease models, and found that they could boost neurite outgrowth, protect neurons, and promote neural progenitor proliferation. Our study highlights the utility of secretome profiling of iAst and demonstrates its applications in disease study and target identification and validation in drug development.

neuroscience↗