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Stubbert, C. A.

Publications and source records attributed to Stubbert, C. A..

2 recordsLinked to original sources

Long-term ex ovo culture of Caenorhabditis elegans embryos

While the genetic tractability, transparency and invariant development of the Caenorhabditis elegans embryo have led to its broad adoption as a model system for the study of cell and developmental biology, its impermeable eggshell has complicated the use of small-molecule reagents during embryogenesis. Existing genetic approaches for rendering the embryo permeable to acute small molecule treatment have increased the accessibility of early embryogenesis to pharmacological manipulation but compromise long-term viability, preventing their use in studies of later developmental processes or post-exposure physiology. Here, we describe the use of an optimized enzymatic eggshell digestion protocol coupled with a minimal, serum-free culture medium that supports the survival and normal development of ex ovo embryos through larval maturation and adulthood. We show that this approach renders embryos permeable to a wide range of small molecules, enabling precise temporal manipulation of developmental processes previously inaccessible through conventional genetic methods. We demonstrate the utility of this technique through the pharmacological modulation of cytoskeletal components including microtubules and actin, as well as the minus-end-directed microtubule motor protein dynein, highlighting applications for the study of cell division, morphogenesis, and neuronal development, especially at later stages of embryogenesis. As a proof of concept, we use acutely timed dynein inhibition to show that cytoplasmic dynein is required to transport the centriole into the dendrite of embryonically born sensory neurons. This approach expands the experimental toolkit available for labeling and manipulating developmental processes in C. elegans.

cell biology↗

Axis reset is rate limiting for onset of whole-body regenerative abilities during planarian development

Few studies have investigated whether or how regenerative abilities vary across developmental stages of animal life cycles. Determining mechanisms that promote or limit regeneration in certain life cycle stages may pinpoint the most critical factors for successful regeneration and suggest strategies for reverse-engineering regenerative responses in therapeutic settings. In contrast to many mammalian systems, which typically show a loss of regenerative abilities with age, planarian flatworms remain highly regenerative throughout adulthood. The robust reproductive and regenerative capabilities of the planarian Schmidtea polychroa (Spol) make them an ideal model to determine when and how regeneration competence is established during development. We report that Spol gradually acquires whole body regenerative abilities during late embryonic and early juvenile stages. Posterior (tail) regenerative abilities are constitutive, whereas anterior (head) regenerative abilities are dependent on developmental stage, tissue composition of the amputated fragment, and axial position of the cut plane. Stem-like cells are required, but not sufficient, for onset of head regeneration ability. We propose that regulation of main body axis reset, specifically the ability to remake an anterior organizing center, is a rate-limiting factor for establishment of whole-body regeneration competence. Supporting this hypothesis, knock-down of the canonical Wnt pathway effector Spol {beta}-catenin-1, a posterior determinant, induces precocious head regeneration under conditions that are normally head regeneration incompetent. Our results suggest that regeneration competence emerges through interactions between cycling stem-like cells, the cellular source of new tissue, and developing adult tissue(s) harboring axial patterning information.

developmental biology↗