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Schlueter, O. M.

Publications and source records attributed to Schlueter, O. M..

2 recordsLinked to original sources

Contextual unfamiliarity during drug intake induces AMPA receptors linked to relapse vulnerability

Relapse to drug use remains a challenge in treating substance use disorders (SUDs). In rodent models of SUDs, Ca2+-permeable AMPA receptors (CP-AMPARs) in nucleus accumbens (NAc) medium spiny neurons enhance cue-drug memories after prolonged withdrawal from some forms of drug-conditioned training. Inhibition or removal of these receptors dampens drug-seeking behavior. However, it remains unclear which contextual variables associated with drug use govern CP-AMPAR expression and whether these receptors exhibit the long-term persistence expected of a reliable addiction-related biomarker. Here, we compared three fentanyl-conditioning settings in mice: conditioned place preference (CPP), open-field (OF), and home-cage (HC). Contrary to the HC condition, synaptic CP-AMPAR recruitment was observed in the NAc shell of both CPP and OF groups, which involved exposure to an unfamiliar context following drug administration. CPP and OF training produced comparable increases in CP-AMPAR expression, while CPP mice displayed heightened fentanyl-seeking behavior and OF mice showed locomotor sensitization. After more than 100 days of withdrawal, neither CP-AMPAR expression nor fentanyl-seeking behavior was detected following context re-exposure alone. However, fentanyl re-exposure reinstated drug-seeking behavior despite the absence of CP-AMPAR recruitment, which re-emerged only hours after drug administration. These findings indicate that contextual unfamiliarity drives CP-AMPAR recruitment after drug conditioning and suggest that these receptors transiently govern relapse in consecutive synaptic adaptations during withdrawal. The habituation or avoidance of contextual novelty during opioid treatments may therefore represent a previously underappreciated strategy to limit the recruitment of these maladaptive plasticity mechanisms and reduce relapse risk.

neuroscience↗

PSD-95 drives binocular vision maturation critical for predation

Postsynaptic density protein 95 (PSD-95) is a signalling scaffold within the postsynaptic density of excitatory synapses which drives silent synapse maturation during critical periods (CP). Binocularity develops during visual CPs and matures before its closure. Despite lifelong critical period plasticity, PSD-95 knock-out (KO) mice were reported with only subtle sensory phenotypes as adults. To assess PSD-95s role in ethologically relevant binocular visual processing, we compared prey capture behaviour in PSD-95 KO and wild-type (WT) mice. KO mice were profoundly impaired in diverse epochs of predatory behaviour, but exhibited improved prey localisation under monocular conditions, reminiscent of impaired binocular integration. This was confirmed in an orientation discrimination task, where KO mice were impaired binocularly but performed monocularly like WT mice. Furthermore, binocular orientation discrimination was impaired after knocking down PSD-95 in primary visual cortex (V1), while a superior colliculus-specific knock-down had no effect. These results support an essential role of PSD-95 in binocular behaviour, which becomes evident under ethologically demanding behaviours.

neuroscience↗