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Sansare, A. A.

Publications and source records attributed to Sansare, A. A..

2 recordsLinked to original sources

Impaired Sensory Gating During Standing Balance in Parkinson's Disease

While motor deficits in Parkinsons disease (PD) are well-studied, the role of somatosensory processing in postural instability remains unclear. It is unknown whether sensory gating, a mechanism for filtering irrelevant sensory input, is impaired during standing balance in individuals with PD. To address this, we investigated cortical sensory processing in individuals with PD, age-matched older adults (OA), and young adults (YA) as they performed four balance tasks of increasing difficulty. We measured postural sway using a force platform and recorded somatosensory-evoked potentials (SEPs) from the primary somatosensory cortex (S1) following tibial nerve stimulation. Our results showed a clear dissociation between behavior and neurophysiology. Although postural sway was comparable between the PD and OA groups, only the OA and YA groups showed intact sensory gating, with SEP amplitudes decreasing as the balance challenge increased. In contrast, participants with PD demonstrated consistently elevated SEP amplitudes across all conditions. This study provides the first direct evidence of impaired sensory gating during standing balance in PD. These findings indicate a fundamental deficit in the cortical processing of sensory information essential for postural control. Consequently, they underscore the critical need for therapeutic interventions that target sensory integration deficits, not just motor symptoms. Key pointsO_LIHealthy young and older adults demonstrate intact sensory gating during standing balance, with somatosensory-evoked potential (SEP) amplitudes decreasing as postural difficulty increases. C_LIO_LIIndividuals with Parkinsons disease (PD) show impaired sensory gating, with elevated SEP amplitudes that are not appropriately modulated by increasing postural demands. C_LIO_LIDespite comparable postural sway to healthy older adults, the PD group exhibited fundamentally different neurophysiological responses to balance challenges. C_LIO_LIThis dissociation between motor performance and neurophysiology indicates a primary deficit in cortical sensory processing in PD. C_LIO_LIImpaired sensory gating may reflect a key, independent contributor to postural instability in PD, highlighting the need to target sensory deficits in treatment. C_LI

neuroscience↗

Relationships between balance performance and connectivity of motor cortex with primary somatosensory cortex and cerebellum in middle aged and older adults

Connectivity of somatosensory cortex (S1) and cerebellum with the motor cortex (M1) is critical for balance control. While both S1-M1 and cerebellar-M1 connections are affected with aging, the implications of altered connectivity for balance control are not known. We investigated the relationship between S1-M1 and cerebellar-M1 connectivity and standing balance in middle-aged and older adults. Our secondary objective was to investigate how cognition affected the relationship between connectivity and balance. Our results show that greater S1-M1 and cerebellar-M1 connectivity was related to greater postural sway during standing. This may be indicative of an increase in functional recruitment of additional brain networks to maintain upright balance despite differences in network connectivity. Also, cognition moderated the relationship between S1-M1 connectivity and balance, such that those with lower cognition had a stronger relationship between connectivity and balance performance. It may be that individuals with poor cognition need increased recruitment of brain regions (compensation for cognitive declines) and in turn, higher wiring costs, which would be associated with increased functional connectivity.

neuroscience↗