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Salimi, A.

Publications and source records attributed to Salimi, A..

2 recordsLinked to original sources

InterOpt: Improved gene expression quantification in qPCR experiments using weighted aggregation of reference genes

Quantification of gene expression is a crucial task in biomedical studies. Although high-throughput methods enable rapid and simultaneous expression quantification of coding or non-coding regions, qPCR is still vastly used due to its high availability, sensitivity, specificity, reproducibility, low cost, and ease of use. A limitation of qPCR has been the need of internal controls or reference genes (RGs) with stable expression in different conditions, to normalize the expression level of the other target genes. So far, several stability criteria and numerous methods for selecting a group of RGs have been proposed, however, important challenges must be addressed. Here we introduce a mathematical basis for precise modeling of qPCR expression normalization and justify widely used stability measures of RGs. We then propose a family of scale-invariant functions, as an alternative to the geometric mean, to optimize aggregated expression of RGs. We provide closed-form optimizations for several scale-invariant aggregation functions. Among them, we show the superiority of weighted geometric mean, whose parameters optimize standard deviation as the stability measure of aggregated RGs expression. We provide experimental support for this finding using real data of solid tumors and liquid biopsies of different sample sizes. The proposed methods can be easily integrated in the existing qPCR expression normalization pipelines of genes and non-coding RNAs. We also provide an implementation of the proposed methods as an R package, with GPU acceleration. Availability and implementationhttps://github.com/asalimih/InterOpt Contactasalimih@gmail.com

bioinformatics↗

Hand cold pressor test induces thermogenesis in upper thoracic regions as measured by skin surface infrared thermography

BackgroundCold exposure may cause health problems and impaired productivity in outdoor or cold-temperature workers. The cold pressor test (CPT) is a laboratory procedure that measures cardiovascular and thermoregulatory responses to acute cold exposure such as metabolic activity in brown adipose tissue. How the body responds to acute cold exposure of a hand is not completely understood. We tested the hypothesis that the upper thorax produces heat during a single hand-CPT, which restores warmth to the cold-expose appendage. ObjectivesThe objective was to measure skin temperature changes in the upper thoracic regions and the cold-exposed appendage during a CPT. The secondary objective was to determine if cardiovascular or psychological responses during CPT accounted for skin temperature changes. Methods50 healthy participants immersed their right hand up to wrist level in 4 {degrees}C water for three minutes. Surface skin temperatures were imaged by infrared thermography at baseline, during CPT, and in the recovery phase. Sublingual oral temperature and water bath temperature were recorded throughout the test. Cardiovascular responses were monitored by continuous finger pulse-wave plethysmography. Peak pain and peak stress were reported by the participants on a Likert scale. ResultsCPT increased the systolic blood pressure (+22 mmHg, p < 0.001), diastolic blood pressure (+15 mmHg, p < 0.001) and heart rate (+7 beats per minute, p = 0.024). During CPT, skin temperature increased on thoracic regions including mediastinal (+0.5{degrees}C, p < 0.021), sternal (+0.5{degrees}C, p < 0.002), right supraclavicular (+0.3{degrees}C, p < 0.042) and left supraclavicular (+0.3{degrees}C, p < 0.016) regions. During CPT, the hand was cooler on ventral (-14.6{degrees}C p < 0.001) and dorsal (-15.2{degrees}C p < 0.001) sides, and warmed up during recovery. The ventral forearm, dorsal forearm, antecubital fossa, and adjacent medial epicondyle region were significantly cooler throughout the recovery time. The oral temperature did not change during CPT. There were no correlations between the change in mediastinal skin temperature and the sex of the participant, or changes in cardiovascular parameters, peak pain, or peak stress values. ConclusionsLocalized hand cooling caused a rapid warming of the thorax, dissipation of cold in the forearm, and rewarming of the hand during recovery. Thermoregulation was not dependant on pain, stress, sex, or cardiovascular changes between participants. By understanding thermoregulation, better approaches can be developed to mitigate the negative impacts of cold exposure.

physiology↗