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Richker, H.

Publications and source records attributed to Richker, H..

2 recordsLinked to original sources

A Systematic Review of Evidence for the Cost of Therapeutic Resistance in Cancer

Emergence of therapeutic resistance is a critical clinical challenge in cancer treatment, contributing to treatment failure, disease relapse, and patient mortality. Prior to treatment initiation, therapy-resistant clones typically make up such a small proportion of the neoplasm that they can be hard to detect and only expand under the selective pressure of therapy. This suggests that resistant clones incur a fitness cost for resistance to therapy. Whether therapeutic resistance incurs a fitness cost has important clinical implications, including the potential to leverage competition with sensitive cells to prevent the expansion of the resistant population. However, the fitness compromises associated with treatment resistance in cancer are not well-understood at present. We conducted a systematic review for peer-reviewed papers that fulfilled the following selection criteria: (i) experiments depict direct competition, (ii) between therapy-resistant and therapy-sensitive clones, (iii) in an untreated environment. We found 49 experiments that matched those criteria, of which approximately two-thirds found a fitness cost to resistance in a competitive environment. Of all pooled features, we found that the resistance characteristic was most significantly associated with whether resistant clones exhibited a fitness advantage in competition (p=0.0154). Further, we identify complex ecological interactions that may influence the behavior of the cancer cell population in the untreated environment. Predicting which resistance characteristics can be exploited therapeutically and identifying potential methods of modulating the costliness of the resistant phenotype may be critical to future improvements in cancer therapy. Lay SummaryWhen cancer cells evolve to no longer respond to a cancer therapy, do they pay a cost for that invulnerability? We reviewed experiments where cancer cells resistant to a therapy were pitted against cancer cells that were still sensitive to the therapy. Approximately [2/3] of the experiments showed that the sensitive cells could out-compete the resistant cells, implying that the latter were paying some cost for their ability to resist the therapy.

cancer biology↗

Leveraging Comparative Phylogenetics for Evolutionary Medicine: Applications to Comparative Oncology

Comparative phylogenetics provides a wealth of computational tools to understand evolutionary processes and their outcomes. Advances in these methodologies have occurred in parallel with a surge in cross-species genomic and phenotypic data. To date, however, the majority of published studies have focused on classical questions in evolutionary biology, such as speciation and the ecological drivers of trait evolution. Here, we argue that evolutionary medicine in general, and our understanding of the origin and diversification of disease traits in particular, would be greatly expanded by a wider integration of phylogenetic comparative methods (PCMs). We use comparative oncology - the study of cancer across the tree of life - as a case study to demonstrate the power of the approach and show that implementing PCMs can highlight the mode and tempo of the evolutionary changes in intrinsic, species-level disease vulnerabilities.

evolutionary biology↗