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Rees, D. J.

Publications and source records attributed to Rees, D. J..

3 recordsLinked to original sources

Identification and characterization of a wet adhesive protein extracted from Dreissena bugensis, the freshwater quagga mussel

Mechanisms of wet adhesion have been developed by several aquatic organisms over millions of years of evolutionary processes. Yet, the repertoire of synthetic biocompatible wet adhesive materials is still limited. In most marine bioadhesive proteins, 3,4-dihydroxyphenylalanine (DOPA) plays a significant role in strong interfacial interactions. The bioadhesive proteins in freshwater organisms are less well understood. The quagga mussel (Dreissena bugensis) is a notorious freshwater invasive species in the Great Lakes that attaches to a plethora of surfaces via a byssus. To determine the adhesive proteins in the quagga mussel byssus, we utilized quantitative proteomics to identify the proteins enriched at the byssus-substrate interface. Among the identified proteins was the Dbfp7 protein family. Dbfp7 is a small, polymorphic, and mostly disordered protein that lacks significant amounts of DOPA. Atomic force microscopy measurements of Dbfp7 confirm that this protein has similar adhesion energy to marine mussel adhesive proteins in aqueous conditions despite lacking DOPA. These results suggest that freshwater mussels may employ different mechanisms of adhesion compared to marine byssates. The inclusion of Dbfp7 to the library of known wet bioadhesive proteins - the first functionally characterized freshwater bioadhesive protein to our knowledge - will allow for a better understanding of the fundamental properties required to achieve biocompatible wet adhesion, a crucial step for the development of bio-inspired wet adhesive materials, such as improved medical adhesives. Significance StatementWhile many aquatic organisms evolved strategies to adhere to surfaces underwater, current synthetic biocompatible adhesives lack reliable adhesive strength in varying aqueous conditions. Investigating freshwater mussel adhesive proteins expands the current understanding of aquatic bioadhesion and offers potential advancements in wet adhesive technology. The discovery of a DOPA-deficient wet adhesive protein derived from the invasive quagga mussels presents a paradigm shift to the currently known mechanisms of byssal bioadhesion, thereby expanding the repertoire of wet adhesive strategies. Further, understanding the mechanism of adhesion employed by this biofouling species can help with the design of anti-fouling strategies to mitigate the impacts of these animals on the infrastructure and ecosystems in the Great Lakes and connected freshwater systems in North America.

biochemistry↗

Acyl ghrelin attenuates neurochemical and motor deficits in the 6OHDA model of Parkinsons disease

The feeding-related hormone, acyl-ghrelin, protects dopamine neurons in murine 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP)-based models of experimental Parkinsons disease (PD). However, the potential protective effect of acyl-ghrelin on substantia nigra pars compacta (SNpc) dopaminergic neurones and consequent behavioural correlates in the more widely used 6-hydroxydopamine (6-OHDA) rat medial forebrain bundle (MFB) lesion model of PD are unknown. To address this question, acyl-ghrelin levels were raised directly by mini-pump infusion for 7-days prior to unilateral injection of 6-OHDA into the MFB with assessment of amphetamine-induced rotations on days 27 and 35, and immunohistochemical analysis of dopaminergic neurone survival. While acyl-ghrelin treatment was insufficient to elevate food intake or body weight, it attenuated amphetamine-induced circling behaviour and SNpc dopamine neurone loss induced by 6-OHDA. These data support the notion that elevating circulating acyl-ghrelin may be a valuable approach to slow or impair progression of neurone loss in PD. HighlightsO_LIAcyl-ghrelin attenuates SNpc dopamine cell loss in rat 6-OHDA-lesion model of PD C_LIO_LIAcyl-ghrelin attenuates motor deficits in rat 6-OHDA-lesion model of PD C_LI

neuroscience↗

Automated Quantification of Mitochondrial Fragmentation in an In-Vitro Parkinson's Disease Model.

Neuronal mitochondrial fragmentation is a phenotype exhibited in models of neurodegeneration such as Parkinsons Disease. Delineating the dysfunction in mitochondrial dynamics found in diseased states can aid our understanding of underlying mechanisms for disease progression and possibly identify novel therapeutic approaches. Advances in microscopy and the availability of intuitive open-access software has accelerated the rate of image acquisition and analysis, respectively. These developments allow routine biology researchers to rapidly turn hypotheses into results. In this protocol, we describe the utilisation of cell culture techniques, high-content imaging (HCI), and subsequent open-source image analysis pipeline for the quantification of mitochondrial fragmentation in the context of an in-vitro Parkinsons Disease model.

neuroscience↗