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Biology subjects

Paplauskas, S.

Publications and source records attributed to Paplauskas, S..

3 recordsLinked to original sources

Borrowing data from other populations to forecast epidemic size

Forecasting infectious disease dynamics is important for ecological management and emerging disease preparedness, yet many systems lack the long-term datasets required to develop reliable predictions. Spatial replication may provide an alternative by allowing information to be shared among populations, but the extent to which this improves forecasting remains unclear. Here, I used epidemic and temperature data from 20 replicated semi-natural Daphnia-parasite pond populations monitored across four seasons (80 epidemics) to test whether information from multiple populations improves forecasts and whether environmental similarity identifies informative populations. I forecasted disease prevalence, infected host density, and healthy host density using a benchmark model, autoregressive integrated moving average (ARIMA), and time-series regression models. Models were trained using focal-population data alone, mean dynamics across other populations, or temperature-weighted mean dynamics from environmentally similar populations. Epidemics showed strong seasonal structure, with prevalence and infected host density generally peaking during the warmest period of the season. However, no forecasting approach consistently outperformed others across all variables. Temperature improved forecasts of prevalence and infected host density but not healthy host density. Multi-population training improved forecasts in some cases, particularly for ARIMA models, whereas temperature-weighted averaging provided no additional benefit over simple averaging. Regression models consistently produced the most accurate forecasts of healthy host density. These results show that borrowing information among populations can improve forecasts under specific conditions but is not universally beneficial, highlighting the importance of matching forecasting approaches and data sources to the ecological processes being predicted.

ecology↗

Epidemic size and host-parasite (co)-evolution: a meta-analysis

The size of an epidemic can influence the strength of selection acting on hosts and parasites, but whether epidemic size consistently shapes host-parasite (co)-evolution remains unclear. I conducted a meta-analysis of studies that simultaneously quantified epidemic size and evolutionary change in hosts, parasites or their interactions, synthesising 123 effect sizes from 11 studies. I tested whether epidemic size was associated with four components of evolutionary change: host evolution, parasite evolution, non-additive host-parasite coevolution and net coevolution. Larger epidemics were associated with greater host evolutionary change in invertebrates, whereas the relationship was positive in plants and negative but non-significant in bacteria. Parasite evolution, non-additive coevolution and net coevolution were not detectably associated with epidemic size, although parasite infectivity showed a relatively consistent increase across studies. These results provide limited evidence for a general relationship between epidemic size and host-parasite (co)-evolution and instead suggest that evolutionary responses depend on the host-parasite system and broader ecological and temporal context. Interpretation is constrained by the small and taxonomically biased evidence base, particularly the strong representation of Daphnia, and by substantial heterogeneity in how epidemic size and evolutionary change were measured and defined. More studies across diverse host-parasite systems are required that track both evolutionary change and disease prevalence through time. This work also provides an empirical basis for incorporating coevolutionary feedbacks into quantitative epidemiological models.

evolutionary biology↗

The effect of host population genetic diversity on variation in metrics of parasite success

According to conventional wisdom, disease transmission rate is usually higher in more genetically homogenous host populations. Previous studies have principally considered how host population genetic diversity impacts the mean of parasite infection performance. However, when considering risks from epidemics and the emergence of novel infectious diseases, variability in parasite success may be just as important as the mean. Here we propose an Epidemic Diversity Model for how host-parasite population genetic diversity influences both the mean and variability in parasite success. We evaluate this conceptual model by re-analysing effect size data from two meta-analyses, including 211 comparisons of high versus low genetic diversity host populations from 48 studies. Our analysis challenges previous understanding by demonstrating that high host population genetic diversity only reduces mean parasite success for specialist parasites with narrow host range, but not for generalist multi-host parasites. We also find that the combination of host range and parasite population genetic diversity determine the effect of host population genetic diversity on the variability in parasite success. These results have important implications for the management of host population genetic diversity for natural populations.

ecology↗