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Nocera, G.

Publications and source records attributed to Nocera, G..

2 recordsLinked to original sources

Long-lasting structural and functional maturation in transplanted human neurons reprogrammed from glial progenitor cells

Direct reprogramming of brain-resident glial progenitor cells (GPCs) into induced neurons (iN) shows great promise for future regenerative therapies for brain diseases. This type of cell conversion is facilitated by introducing lineage-specific transcription factors or small molecules to somatic cells, and can even be achieve in vivo, targeting resident glia cells in the brain for in vivo conversion. While this have shown promising results in animal models, a crucial step is to show the long-term survival and maturation of human reprogrammed cells in vivo. However, this remains largely unexplored partly due to the inaccessible cell source of GPC and difficulty in transplanting converted neurons. In this study, we assessed human GPCs derived iNs for their long-term structural and functional neuronal maturation after transplantation to the immunodeficient mouse brain. GPCs were transduced with established transcription factor cocktail and transplanted to the medial prefrontal cortex as three-dimensional cultures to improve survival and integration upon transplantation. Our results reveal survival of hiNs, for up to 10 months post-transplantation (MPT) and wide distribution across local and proximal functionally connected brain regions. Morphological analysis revealed a gradual neuronal maturation over time, characterized by increase in cellular complexity and expression of neuronal markers. Supporting these findings, electrophysiological recordings demonstrated progressive functional maturation up to 5 MPT with increase in sodium and potassium currents, and the ability to generate multiple evoked and spontaneous action potentials, validating a neuronal phenotype. Importantly, these changes were absent in control grafts of non-converted hGPCs that retained their glia identity. Our findings indicate that hGPCs derived iNs maintain a stable neuronal phenotype that persist in the mouse brain for extended periods. These cells exhibit survival and remarkable adaptability to the host environment, enabling both structural and functional maturation over time. These results highlight their potential as a promising strategy for neural replacement therapies aimed at restoring damaged circuits and promoting brain repair. Highlights* Human neurons reprogrammed from glia progenitor cells survive long term upon transplantation in the immunodeficient mouse brain. * The human induced neurons migrate to local and distal connected brain regions within prefrontal cortex circuitry. * Grafted neurons gradually develop neuronal complex morphology and electrophysiological functional properties in the mouse brain.

neuroscience↗

Repair oligodendrocytes demyelinating and disintegrating damaged axons after injury

After a spinal cord injury, axons fail to regrow, which results in permanent loss of function1. This is in contrast with peripheral axons that can regrow efficiently after injury2. These differences are partly due to the different plasticity of myelinating cells, Schwann cells and oligodendrocytes, in these two systems3. The molecular mechanisms underlying this different plasticity remain however poorly understood. Here, we show that the phosphatase Dusp64 is a master inhibitor of oligodendrocyte plasticity after spinal cord injury. Dusp6 is rapidly downregulated in Schwann cells and upregulated in oligodendrocytes after axon injury. Simultaneously, the MAP kinases ERK1/2 are activated and the transcription factor c-Jun is upregulated in Schwann cells5,6, but not in oligodendrocytes. Ablation or inactivation of Dusp6 induces rapid ERK1/2 phosphorylation, c-Jun upregulation and filopodia formation in oligodendrocytes, leading to mechanically-induced, fast disintegration of distal ends of injured axons, myelin clearance and axonal regrowth. Together, our findings provide understanding of the mechanisms underlying the different plasticity of Schwann cells and oligodendrocytes after injury and a method to convert mature oligodendrocytes exhibiting inhibitory cues for axonal regrowth into repair oligodendrocytes reminiscent of repair Schwann cells. We show that repair oligodendrocytes successfully increase the compatibility of the spinal cord environment with axonal regrowth after injury, suggesting a potential use of repair oligodendrocytes as future therapeutic approach to treat spinal cord injuries.

neuroscience↗