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Marino, D.

Publications and source records attributed to Marino, D..

2 recordsLinked to original sources

DEGoldS: a workflow to assess the accuracy of differential expression analysis pipelines through gold-standard construction

RNA sequencing (RNA-seq) is a high throughput sequencing method that has become one the most employed tools in transcriptomics. The implementation of optimal bioinformatic analyses required in RNA-seq experiments may be complicated due to the large amounts of data generated by the sequencing platforms, along with the intrinsic nature of these data types. In the last years many programs and pipelines have been developed for differential expression (DE) analyses, but their effectiveness can be reduced when working with non-model species lacking public genomic resources. Moreover, there is not a universal recipe for all the experiments and datasets and the modification of standard RNA-seq bioinformatic pipelines through parameter tuning and the use of alternative software may have a strong impact in the outcome of DE analysis. Therefore, although the selection of the most accurate DE pipeline configuration and the evaluation of how these changes could affect the final DE results in RNA-seq experiments is mandatory to reduce bias, the lack of gold-standard datasets with known expression patterns hampers its implementation. In the present manuscript we present DEGoldS, a workflow consisting on sequential Bash and R scripts to construct gold-standards for simulation-based benchmarking of user selected pipelines for DE analysis and the computation of the accuracy of the pipelines. We validated the workflow with a case study consisting on real RNA-seq libraries of radiata pine, an important forest tree species with no publicly available reference genome. The results showed that slight pipeline modifications produced remarkable differences in the outcome of DE analysis.

bioinformatics↗

The Actin Bundling Protein Fascin Promotes Lung Cancer Growth and Metastasis by Enhancing Glycolysis and PFKFB3 Expression

Fascin is a pro-metastasis actin bundling protein upregulated in essentially all the metastatic carcinoma. It is believed that fascin promotes cancer cell migration and invasion by facilitating membrane protrusions such as filopodia and invadopodia. Aerobic glycolysis is a key feature of cancer metabolism that provides critical intermediate metabolites for tumor growth and cell proliferation. Here we report that fascin increase glycolysis in lung cancer to promote tumor growth and metastasis. Fascin promotes glycolytic flux by increasing the expression and activities of phosphofructose kinase 1 and 2 (PFK1 and 2). The glycolytic function of fascin depends on activation of YAP1 through its canonical actin bundling activity. Fascin promotes the binding of YAP1 to a TEAD1/4 binding motif located 30 bp upstream of the PFKFB3 transcription start site to activate its transcription. Our interrogation of the TCGA database suggest that the fascin-YAP1-PFKFB3 circuit is likely conserved across different types of cancer. We further showed that the glycolytic function of fascin is essential for promotion of lung cancer growth and metastasis. Importantly, pharmacological inhibitors of fascin could be used to suppress the YAP1-PFKFB3 signaling and inhibit glycolysis in cancer cell lines, organoid cultures and xenograft metastasis models. Taken together, our data reveal an important glycolytic role of fascin in lung cancer metabolism, and suggest that pharmacological inhibitors of fascin could be used to reprogram cancer metabolism in lung cancer and potentially other cancer with fascin upregulation.

cancer biology↗