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Jerlhag, E.

Publications and source records attributed to Jerlhag, E..

2 recordsLinked to original sources

Tirzepatide attenuates dopamine reward signaling and suppresses alcohol drinking and relapse-like behaviors in rodents

Alcohol use disorder (AUD) remains a major public health problem, with few effective medications currently available. However, peptides of the gut-brain axis appear to offer promising therapeutic targets for AUD as they influence the mesolimbic reward circuitry. Here, we examined the effects of tirzepatide, a long-acting dual glucagon-like peptide-1 receptor (GLP-1R) and glucose-dependent insulinotropic polypeptide receptor (GIPR) agonist approved for diabetes and obesity, using behavioral assays, alcohol intake paradigms, and molecular analyses in rodents. First, tirzepatide effectively attenuated the rewarding properties of alcohol, measured through locomotor stimulation, conditioned place preference, and accumbal dopamine release. Subsequently, this GLP-1R/GIPR agonist dose-dependently reduced voluntary alcohol consumption, prevented binge and relapse-like drinking, and maintained efficacy during repeated administration. Finally, tirzepatide induced sustained synaptic depression in the lateral septum and further altered histone regulatory proteins in this region, suggesting a potential neural substrate for its effects. Moreover, the GLP-1R/GIPR agonist affected metabolic parameters including body weight, adipose tissue mass, hepatic triglycerides and circulating pro-inflammatory cytokines. Together, our findings suggest tirzepatide modulates alcohol-related behaviors through reward-related mechanisms while also affecting physiological consequences associated with long-term alcohol use. Given tirzepatides established clinical use and the consistency of effects observed here, these results support further investigation for treating AUD and associated complications. SIGNIFICANCE STATEMENTExisting treatments for alcohol use disorder show limited effectiveness, leaving patients without viable therapeutic options. We demonstrate that tirzepatide, a long-acting gut peptide-based drug already approved for diabetes and obesity, substantially reduces alcohol consumption and prevents relapse-like behavior across multiple preclinical models. Tirzepatide appears to work by influencing brain reward systems while simultaneously affecting metabolic complications common in alcohol disorders. Given tirzepatides clinical availability, these findings suggest repurposing a recently approved drug to tackle one of medicines more persistent treatment challenges.

neuroscience↗

Decoding the Influence of Central LEAP2 on Hedonic Food Intake and its association with Dopaminergic Reward Pathways

The gut-brain peptide ghrelin and its receptor (GHSR) are established as a regulator of hunger and reward-processing. However, the recently recognized GHSR inverse agonist, liver-expressed antimicrobial peptide 2 (LEAP2), is less characterized. Given the role of GHSR in many central processes, and in particular reward, understanding the central effects of LEAP2 is of high interest to understand reward-related behaviors and disorders, including hedonic feeding in eating disorders. The present study aimed to elucidate LEAP2s central effect on reward-related behaviors through hedonic feeding and its mechanism. LEAP2 was administrated centrally in male mice and effectively reduced hedonic feeding but had no or little effect on homeostatic chow intake when a more palatable option was available. Strikingly, the effect on hedonic feeding was correlated to the preference of the palatable food option, where peanut butter showed the highest preference and the greatest reduction by LEAP2. Further, LEAP2 reduced the rewarding memory of high-preference foods, as well as attenuated the accumbal dopamine release associated with peanut butter exposure and eating. Interestingly, LEAP2 was widely expressed in the brain, and in particular in reward-related brain areas such as the laterodorsal tegmental area (LDTg). The expression in this area was also markedly altered when given free access to peanut butter. Accordingly, infusion of LEAP2 into the LDTg was sufficient to attenuate acute peanut butter eating. Taken together, the present results show that central LEAP2 has a profound effect on central dopaminergic reward signaling and affects several aspects of hedonic eating. The present study highlights LEAP2s effect on reward, which may have application not only for hedonic feeding, but for other reward-related psychiatric disorders as well.

neuroscience↗