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Hoefler, T.

Publications and source records attributed to Hoefler, T..

2 recordsLinked to original sources

Atlas-independent brain connectome analysis at voxel-level granularity: graph convolutional networks for etiology classification in newborns

Early identification of altered brain networks in neonates at risk for neurodevelopmental impairments is critical for timely intervention and improving outcomes. This study explores the potential of graph convolutional networks (GCNs) applied to structural brain connectomes at the voxel level granularity to classify neonatal connectomes by their underlying etiology: 51 children with congenital heart disease (CHD), 100 children born very preterm (PB), and 43 children with spina bifida aperta (SBA). Leveraging the flexibility of voxel-level parcellation, we captured fine-grained connectomic differences that improved classification performance (F1 = 0.78) compared to both atlas-based methods (F1 = 0.62) and a multilayer perceptron baseline model (F1 = 0.69). This approach enables subject-specific parcellation without the need for predefined anatomical templates, facilitating the analysis of diverse brain morphologies and age ranges. Attribution analysis using integrated gradients provided interpretable insights into etiology-specific connectomic patterns, highlighting regions of potential neurodevelopmental importance, such as the Rolandic operculum, inferior parietal lobule, and inferior frontal gyrus. Lateralized attribution patterns in PB reflected known neurodevelopmental alterations, underscoring the value of interpretable graph learning for understanding etiology-specific connectomic features. This work represents an important step toward atlas-independent connectome analysis, offering a novel framework for studying diverse neonatal populations and advancing our understanding of early brain development.

neuroscience↗

Neural Network-Assisted Humanization of COVID-19 Hamster scRNAseq Data Reveals Matching Severity States in Human Disease

Translating findings from animal models to human disease is essential for dissecting disease mechanisms, developing and testing precise therapeutic strategies. The coronavirus disease 2019 (COVID-19) pandemic has highlighted this need, particularly for models showing disease severity-dependent immune responses. Single-cell transcriptomics (scRNAseq) is well poised to reveal similarities and differences between species at the molecular and cellular level with unprecedented resolution. However, computational methods enabling detailed matching are still scarce. Here, we provide a structured scRNAseq-based approach that we applied to scRNAseq from blood leukocytes originating from humans and hamsters affected with moderate or severe COVID-19. Integration of COVID-19 patient data with two hamster models that develop moderate (Syrian hamster, Mesocricetus auratus) or severe (Roborovski hamster, Phodopus roborovskii) disease revealed that most cellular states are shared across species. A neural network-based analysis using variational autoencoders quantified the overall transcriptomic similarity across species and severity levels, showing highest similarity between neutrophils of Roborovski hamsters and severe COVID-19 patients, while Syrian hamsters better matched patients with moderate disease, particularly in classical monocytes. We further used transcriptome-wide differential expression analysis to identify which disease stages and cell types display strongest transcriptional changes. Consistently, hamsters response to COVID-19 was most similar to humans in monocytes and neutrophils. Disease-linked pathways found in all species specifically related to interferon response or inhibition of viral replication. Analysis of candidate genes and signatures supported the results. Our structured neural network-supported workflow could be applied to other diseases, allowing better identification of suitable animal models with similar pathomechanisms across species. Key PointsO_LINeural networks can successfully match disease states between animal models and humans using single-cell data as shown for COVID-19 C_LIO_LIModerately diseased patients best matched Syrian hamster cells; severely diseased patients best matched Roborovski hamster neutrophils C_LI

immunology↗