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Hegde, R. S.

Publications and source records attributed to Hegde, R. S..

2 recordsLinked to original sources

A unified evolutionary origin for SecY and YidC

Cells use transporters to move protein across membranes, but the origins of the most ancient transporters are unknown. Here, we analyse the ubiquitous protein-conducting channel SecY. Features conserved by its two duplicated halves suggest that their common ancestor was an antiparallel homodimeric channel. Structural searches with SecYs halves detect exceptional similarity with the only other ubiquitous protein transporter, YidC. Their shared fold comprises a three-helix bundle interrupted by a helical hairpin. In YidC this hairpin is cytoplasmic and facilitates substrate delivery, whereas in SecY it is transmembrane and forms the substrate-binding lateral gate helices. In both, the three-helix bundle forms a protein-conducting hydrophilic groove, delimited by a conserved hydrophobic residue. We propose that SecY originated as a homodimeric YidC homolog. Many YidC homologs now use this interface to heterodimerise with a conserved partner. Unification of the two ubiquitous protein transporters would reconstruct a key step in the evolution of cells.

molecular biology

The architecture of EMC reveals a path for membrane protein insertion

Approximately 25% of eukaryotic genes code for integral membrane proteins that are assembled at the endoplasmic reticulum. An abundant and widely conserved multi-protein complex termed EMC has been implicated in membrane protein biogenesis, but its mechanism of action is poorly understood. Here, we define the composition and architecture of human EMC using biochemical assays, crystallography of individual subunits, site-specific photocrosslinking, and cryo-EM reconstruction. Our results show that EMCs cytosolic domain contains a large, moderately hydrophobic vestibule that binds a substrates transmembrane domain (TMD). The cytosolic vestibule leads into a lumenally-sealed, lipid-exposed intramembrane groove large enough to accommodate a single substrate TMD. A gap between the cytosolic vestibule and intramembrane groove provides a path for substrate egress from EMC. These findings suggest how EMC facilitates energy-independent membrane insertion of TMDs, explain why only short lumenal domains are translocated by EMC, and constrain models of EMCs proposed chaperone function.

biochemistry