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Gerassy-Vainberg, S.

Publications and source records attributed to Gerassy-Vainberg, S..

3 recordsLinked to original sources

CytoPro: A computational platform for accurate and robust assessment of cell contributions in bulk expression from diverse tissues and conditions

Cells are the quanta unit of biology and their relative composition in a tissue is the prime driver of bulk tissue gene expression variation. When there is no cell information, deconvolution is an effective tool to achieve cell resolution, which provides important information for learning disease complexity and its interactions with treatments, drugs and/or the environment in a wide variety of contexts. Here we present CytoPro, a production-level tissue and condition-specific deconvolution platform, based on a large collection of human tissue-specific signatures derived from single and sorted cells. CytoPro infer per-sample multiple cell-type composition, given input bulk gene expression. CytoPro includes a rigorous QC pipeline for learning, generating and selecting signatures and performs internal automated validation using multiple QC test criteria including: Comparison to ground truth cytometry and pure sorted cells data, performance evaluation using simulated data including robustness to noise as well as agreement with biological expectations in validation datasets regarding genes and cells. We demonstrate that CytoPro outperforms existing deconvolution tools, in both accuracy and robustness. By exploring multiple datasets with predefined disease phenotypes, and analyzing a use-case of biological treatment response, we show the ability of CytoPro to flush out relevant cell biology in real pathological conditions.

bioinformatics

A personalized network framework reveals predictive axis of anti-TNF response across diseases

Personalized treatment of complex diseases has been mostly predicated on biomarker identification of one drug-disease combination at a time. Here, we used a novel computational approach termed Disruption Networks to generate a new data type, contextualized by cell-centered individual-level networks, that captures biology otherwise overlooked when performing standard statistics. The new data-type extends beyond the feature level space, to the relations space, by quantifying individual-level breaking or rewiring of cross-feature relations. Applying disruption network to dissect high-dimensional blood data, we discover and validate that the RAC1-PAK1 axis is predictive of anti-TNF response in inflammatory bowel disease. Intermediate monocytes, which correlate with the inflammatory state, play a key role in the RAC1-PAK1 responses, supporting their modulation as a therapeutic target. This axis also predicts response in rheumatoid arthritis, validated in three public cohorts. Our findings support blood-based drug response diagnostics across immune-mediated diseases, implicating common mechanisms of non-response.

systems biology

Intestinal dysbiosis in carbapenem-resistant Enterobacteriaceae carriers

Infection with Carbapenem-Resistant Enterobacteriaceae (CRE) became an important challenge in health-care settings and a growing concern worldwide. Since infection is preceded by colonization, an understanding of the latter may reduce CRE-infections. We aimed to characterize the gut microbiota after colonization by CRE, assuming that an imbalanced gastrointestinal tract (GIT)-associated microbiota precedes CRE-colonization. We evaluated the GIT-microbiota using 16S rRNA genes sequencing extracted of fecal samples, collected from hospitalized CRE-carriers, and two control groups of hospitalized non-carriers and healthy adults. The microbiota diversity and composition in CRE-colonized patients differed from that of the control groups participants. These CRE-carriers displayed lower phylogenetic diversity and dysbiotic microbiota, enriched with members of the Enterobacteriaceae family. Concurrent with the bloom in Enterobacteriaceae, a depletion of anaerobic commensals was observed. Additionally, changes in several predicted metabolic pathways were observed for the CRE-carriers. Concomitant, we found higher prevalence of bacteremia in the CRE-carriers. Several clinical factors that might induce change in the microbiota were examined and found as insignificant between the groups. CRE-colonized patients have dysbiotic gut microbiota in terms of diversity and community membership, associated with increased risk for systemic infection. Our study results provides justification for attempts to restore the dysbiotic microbiota with probiotics or fecal transplantation.

microbiology