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Cerquetella, C.

Publications and source records attributed to Cerquetella, C..

2 recordsLinked to original sources

Human single-neuron recordings reveal population coding of attentional dynamics during naturalistic movie viewing

Our eyes move constantly. Where they land is shaped by internal cognitive state, making eye movements a rare, non-invasive window onto that state. However, how moment-to-moment eye-movements map onto human neural population activity during naturalistic viewing remains poorly characterized. We analyzed a public intracranial dataset in which 14 neurosurgical patients watched an eight-minute movie during simultaneous eye-tracking and neuronal activity recording. Single-neuron activity from 814 neurons across anterior cingulate cortex, pre-supplementary motor area, amygdala, hippocampus, and ventromedial prefrontal cortex was pooled into a single distributed population and related to a joint eye-movement state comprising pupil size, saccade rate and fixation duration. Using a cross-validated, multivariate analysis of population activity we found group-level coupling between the neural population and the joint eye-movement state (mean held-out canonical r = 0.145; one-sample t-test p = 0.0003; 9 of 14 subjects individually significant). The coupling was carried principally by saccade dynamics (r = 0.141) and pupil size (r = 0.094), with fixation duration contributing only marginally (r = 0.041). At the single-neuron level the picture differed: more neurons were eye-coupled than expected by chance, yet so weakly that almost none survived correction for multiple comparisons. Read out jointly, the same weakly coupled cells cohered into one reliable population dimension, and no single region's removal significantly reduced it. This pattern is more consistent with a distributed, redundant organization than with a small set of strongly coupled cells. In summary, eye-tracking offers a non-invasive window onto population-level neural states in humans.

neuroscience↗

Scaling of ventral hippocampal activity during anxiety

The hippocampus supports a multiplicity of functions, with the dorsal region contributing to spatial representations and memory, and the ventral hippocampus (vH) being primarily involved in emotional processing. While spatial encoding has been extensively investigated, how the vH activity is tuned to emotional states, e.g. to different anxiety levels, is not well understood. We developed an adjustable linear track maze (aLTM) for mice with which we could induce a scaling of behavioral anxiety levels within the same spatial environment. Using in vivo single-unit recordings, optogenetic manipulations and the application of a convolutional classifier, we examined the changes and causal effects of vH activity at different anxiety levels. We found that anxiogenic experiences activated the vH and that this activity scaled with increasing anxiety levels. We identified two processes that contributed to this scaling of anxiety-related activity: increased tuning and successive remapping of neurons to the anxiogenic compartment. Moreover, optogenetic inhibition of the vH reduced anxiety across different levels, while anxiety-related activity scaling could be decoded using a convolutional classifier. Collectively, our findings position the vH as a critical limbic region that functions as an anxiometer by scaling its activity based on perceived anxiety levels. Our discoveries go beyond the traditional theory of cognitive maps in the hippocampus underlying spatial navigation and memory, by identifying hippocampal mechanisms selectively regulating anxiety.

neuroscience↗