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Biology subjects

Ceminsky, M.

Publications and source records attributed to Ceminsky, M..

2 recordsLinked to original sources

The GT1 domain of RNase J ensures RNA quality control through dsRNA binding in Arabidopsis plastids

RNase J is a ribonuclease found in bacteria, archaea, and plant chloroplasts, and plays diverse roles in RNA maturation and stability. Chloroplast RNase J is encoded by the nuclear RNJ locus and is essential for embryo maturation. Arabidopsis or tobacco plants depleted for RNase J accumulate massive amounts of double-stranded RNA, which interferes with translation and causes chlorosis. Land plant RNase J uniquely contains a C-terminal GT1 domain, a DNA- binding motif found in transcription factors. Here, we have used complementation of an Arabidopsis rnj mutant with versions of RNase J with a mutated or deleted GT1 domain to investigate its role in RNase J function. We show that in vitro, the recombinant GT1 domain binds both double-stranded RNA and DNA, but not single-stranded nucleic acids, with no sequence specificity. Furthermore, while RNase J lacking GT1 binding complements the rnj mutant, these plants accumulate high levels of dsRNA as detected by immunolocalization and RNA-Seq. GT1 mutations also change RNase J solubility in vivo, suggesting that the GT1 domain is involved in localization within the plastid. Taken together, our results suggest that the GT1 domain plays a key role in dsRNA removal through localizing the enzyme and/or selectively binding the dsRNA substrate. GRAPHICAL ABSTRACT O_FIG O_LINKSMALLFIG WIDTH=200 HEIGHT=81 SRC="FIGDIR/small/682605v1_ufig1.gif" ALT="Figure 1"> View larger version (21K): org.highwire.dtl.DTLVardef@e5642borg.highwire.dtl.DTLVardef@136456org.highwire.dtl.DTLVardef@1205466org.highwire.dtl.DTLVardef@184f72b_HPS_FORMAT_FIGEXP M_FIG C_FIG

plant biology↗

Dnmt3bas regulates transcriptional induction and alternative splicing of Dnmt3b

During mammalian embryogenesis, DNMT3B activity is critical for the genome-wide establishment of DNA methylation. Using naive ESC differentiation as a model, we elucidated the mechanism by which lncRNA, Dnmt3bas, controls the inducible expression and alternative splicing of Dnmt3b. Our data showed that Dnmt3bas knockdown increased transcriptional induction and decreased H3K27me3 at Dnmt3b cis-regulatory elements post-differentiation. Notably, transcriptional induction of Dnmt3b was accompanied by exon inclusion, switching the major isoform from catalytically inactive Dnmt3b6 to the active Dnmt3b1. While Dnmt3bas overexpression attenuated Dnmt3b induction, it increased the Dnmt3b1:Dnmt3b6 ratio. This observation was explained by a specific interaction of Dnmt3bas with hnRNPL, which promotes exon inclusion. These data suggest that Dnmt3bas coordinates alternative splicing and transcriptional induction of Dnmt3b by facilitating the interaction of hnRNPL and RNA Pol II at the Dnmt3b promoter. This two-pronged mechanism would tightly control DNMT3B activity, ensuring the fidelity and specificity of de novo DNA methylation during development.

molecular biology↗