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Bispo, P.

Publications and source records attributed to Bispo, P..

2 recordsLinked to original sources

Diverging routes of oligodendrocyte recruitment in adaptive and regenerative myelination

Myelination by oligodendrocytes is dynamically regulated throughout life, supporting axon and circuit function during development, activity-dependent plasticity and repair after injury. How new oligodendrocytes are recruited from their lifelong pool of oligodendrocyte precursor cells (OPCs) in these distinct contexts remains unclear. Using high-resolution in vivo imaging in zebrafish, we directly compared activity-induced and demyelination-induced oligodendrogenesis under otherwise matched conditions. Although enhanced motor activity and demyelination both increased oligodendrocyte production, OPCs activated strikingly different transcriptional programmes. Demyelination, but not enhanced activity, induced acute expression of differentiation-associated genes, including the G-protein-coupled receptor Gpr17. Fate-tracking using a Gpr17 knock-in reporter revealed that all newly formed oligodendrocytes arise from Gpr17-positive states. In the healthy CNS, these states emerged exclusively through proliferation-linked OPC divisions, whereas demyelination unlocked a distinct, proliferation-independent conversion of homeostatic OPCs into Gpr17-positive, differentiation-primed cells. Our findings demonstrate the existence of diverging context-dependent routes of OPC recruitment engaged in adaptive and regenerative myelination.

neuroscience↗

Neuronal synapses are stabilised by activity-dependent vesicular release from oligodendrocyte precursor cells

Activity-dependent regulation of synapses has long been attributed to interactions between neurons alone. Here, using in vivo imaging in the zebrafish visual system we show that one type of glia called the oligodendrocyte precursor cell (OPC) closely interacts with pre-synapses of retinal ganglion cell (RGC) axons, and exhibits activity-dependent Vamp2/3-mediated vesicular release. Critically, blocking OPC vesicle release using botulinum toxin, and silencing OPC activity integration through dampening calcium signalling, destabilises RGC pre-synapses. These observations suggest that OPCs provide a non-neuronal pathway for activity-dependent feedback to shape circuit wiring.

neuroscience↗