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Biology subjects

Abdallah, D.

Publications and source records attributed to Abdallah, D..

3 recordsLinked to original sources

Uncovering the simple adhesive strategy of the Toxoplasma parasite for high-speed motility

Toxoplasma gondii is a protozoan parasite that has evolved a developmental morphotype called tachyzoite that navigates between cells and moves in and out of them in a wide repertoire of homeothermic hosts. Relying on a uniquely constant apicobasal bipolarity coupled to an actomyosin-driven retrograde surface flow, the tachyzoite has elaborated a molecular machinery to assemble transient anchoring contacts with the environment, which support the traction force required to power a typical helical gliding motility. Combining micropatterning with live, reflection interference contrast and expansion microscopies, we bring first nanoscale evidence that the tachyzoite needs to build only one apical anchoring contact with the substrate, thus spatially defining a minimal force transmission platform over which it can slide. We uncover that the apicobasal driven surface flow is set up in response to extracellular biochemical cues independent of adhesin release and tachyzoite-surface interactions, hence prior to motile activity. Furthermore, to identify the minimal adhesion requirements for helical gliding at the level of individual molecular species, we combine biochemical and biophysical quantitative assays based on tunable surface chemistry and quartz crystal microbalance with dissipation monitoring. These approaches uncover that glycosaminoglycan (GAG)-parasite interactions are sufficient to promote a productive contact for helical gliding and pave the way for the characterization of the structure and density of the molecules functionally engaged at this essential parasite-substrate mechanosensitive interface.

cell biology↗

Structural Maintenance of Chromosome 3 interacts with the Topoisomerase VI complex and contributes to the oxidative stress response in Arabidopsis thaliana.

In plants adverse environmental conditions can induce the accumulation of reactive oxygen species, such as singlet oxygen or hydrogen peroxide, at the level of the photosynthetic apparatus. The coordinated action of nucleus-encoded genes is required for containing the deleterious effects of reactive oxygen species. The regulation of such genes follows a molecular signalling process between the chloroplast and the nucleus called retrograde signalling. Previously, we proposed that the Topoisomerase VI (Topo VI) complex participates in the singlet oxygen stress response by regulating the expression of specific subsets of nuclear genes. However, the underlying molecular mechanisms remain unresolved. In this study, we demonstrate that the Topo VI subunit BIN4 interacts with the cohesin subunit AtSMC3. We also show that, similarly to Topo VI mutants, a line suppressing AtSMC3 shows constitutive activation of singlet oxygen response genes and enhanced tolerance to photooxidative stress. Together, these results suggest that Topo VI and AtSMC3 control the expression of singlet oxygen response genes and are possibly involved in the acclimation of plants to photooxidative stress conditions.

plant biology↗

Characterization and rescue by oxytocin of an atypical thermo-sensory reactivity in neonatal mice lacking the autism-associated gene Magel2.

Atypical responses to sensory stimuli are considered as a core aspect and early life marker of autism spectrum disorders (ASD). Although recent findings performed in mouse ASD genetic models report sensory deficits, these were explored exclusively during juvenile or adult period. Whether sensory dysfunctions might be present at the early life stage and rescued by therapeutic strategy are fairly uninvestigated. Here we identified that neonatal mice lacking the autism-associated gene Magel2 fail to react to cool sensory stimuli, while autonomic thermoregulatory function is active. This neonatal deficit was mimicked in control neonates by chemogenetic inactivation of oxytocin neurons. Importantly, intranasal administration of oxytocin was able to rescue the phenotype and brain Erk signaling impairment in mutants. This preclinical study establishes for the first-time early life impairments in thermosensory integration and shows the therapeutic potential benefits of intranasal oxytocin treatment on neonatal atypical sensory reactivity.

neuroscience↗